Preclinical research demonstrates that combining the oncolytic virus CF33 with an immune checkpoint inhibitor results in lasting resistance to certain tumors such as colon cancer. City of Hope surgical oncologist Susanne Warner, M.D., senior author of the study, said the virus could be a “gamechanger.”
Posted in biotech/medical, quantum physics, robotics/AI | 1 Comment on H.A.L.O. AI Completes XPRIZE Competition as Finalist with Groundbreaking Unification of Quantum Mechanics and Relativity
P.e.a.c.e!nc. is proud to announce the conclusion as finalists in the $500k Pandemic Response Challenge sponsored by Cognizant with Landmark AI Experiment.
The NHS in England has begun trials of a new miniature camera device that a patient can swallow, to check if they have bowel cancer, as an alternative to traditional colonoscopy.
I didn’t get my 2nd Moderna Vaccine because how sick others became after theirs — nausea, fatigue, headaches for days, lymph nodes the size of a rock that fits in your hand, increased heart rate… Recently a Utah woman died 4 days after her 2nd vaccine-her heart, liver and kidneys failed. Less then a yr ago I read several studies on hospitalized COVID patients — how their kidneys and liver were failing. I read a recent study on how post COVID individuals are now having heart issues. Another study shows how COVID attacks the heart and why such individuals are now having heart issues. In December 2020, 13 individuals died after getting vaccinated (probably more since then). There is a real connection between COVID and organ failure!!!! I wish I kept links of all the information I read. Be happy to find them again. We don’t even know the long term affects of the vaccine — are the vaccinated going to experience long term health issues as well? Take your chances with a vaccine, or not.
A Utah woman in her 30s died four days after receiving the coronavirus vaccine.
Kassidi Kurill, 39, was healthy and happy and “had more energy” than others, according to a KUTV report. Then, four days after she received her second dose of the coronavirus vaccine, she suddenly died.
“She came in early and said her heart was racing and she felt like she [needed] to get to the emergency room,” her family said. Her father, Alfred Hawley, said he woke up to her asking for help.
Many post COVID victims have heart issues. This is why:
A new study has discovered how the SARS-CoV-2 virus attacks and damages the heart, answering a long-standing question about mysterious heart conditions following COVID-19 infection. The results could have large implications on how to effectively treat severe infections and develop new therapies for preventing long-term damage.
Throughout the pandemic, people with severe COVID-19 infection have often displayed symptoms of heart distress. Those with underlying heart conditions are at a greater risk of severe illness if they catch it, and reports of abnormal heart rhythms (arrhythmia) in previously healthy patients with acute COVID-19 have been common.
However, exactly why this happens has eluded scientists until now. Researchers have been unsure whether the heart symptoms are a result of severe inflammation as the body reacts to the infection, or whether the virus particles themselves invade and attack heart cells.
Saving Lives; Changing Minds — Dr. Emanuele Capobianco, MD, Director for Health and Care, International Federation of Red Cross and Red Crescent Societies.
Dr. Emanuele Capobianco, MD, MPH, is the Director for Health and Care at the International Federation of Red Cross and Red Crescent Societies (IFRC), where he leads the IFRC Global Health and Care Team and provides strategic and operational support to 192 National Red Cross and Red Crescent Societies around the world in the areas of community health, emergency health and water/sanitation. He currently also leads the IFRC global response to COVID19 and the IFRC response to the Ebola outbreaks in DRC.
Before this role at IFRC, Dr. Capobianco was the Deputy Executive-Director of The Partnership for Maternal, Newborn & Child Health, a multi-constituency partnership, hosted by the World Health Organization, and is the world’s largest alliance for women’s, children’s and adolescents’ health. He joined there from the Global Fund to Fight AIDS, Tuberculosis and Malaria where he worked as Senior Policy Advisor in the Office of the Executive Director, leading the development of the 2017–2022 Global Fund Strategy.
Dr. Capobianco is a global public health expert with 20 years experience in international institutions at country, regional and headquarters level. He was the Chief of Health and Nutrition in UNICEF Mozambique, where he worked for four years to advance health outcomes for Mozambican children and women.
Previously Dr. Capobianco was at the World Bank as Senior Health Specialist, managing a large portfolio of grants, particularly in South Asia and Eastern Africa. He also worked in Somalia to support the Expanded Program on Immunization, Polio and Tuberculosis programs; and at the WHO Regional Office for the Eastern Mediterranean promoting the STOP TB partnership in the Middle East.
Dr. Capobianco holds a Medical Degree from Pavia University, a Master in Public Health from Johns Hopkins Bloomberg School of Public Health, and a MSc in Humanitarian Assistance from Rome University.
The hack exposed feeds showing the insides of offices, hospitals and businesses, including Tesla.
Electric stimulation may be able to help blood vessels carry white blood cells and oxygen to wounds, speeding healing, a new study suggests.
The study, published in the Royal Society of Chemistry journal Lab on a Chip, found that steady electrical stimulation generates increased permeability across blood vessels, providing new insight into the ways new blood vessels might grow.
The electrical stimulation provided a constant voltage with an accompanying electric current in the presence of fluid flow. The findings indicate that stimulation increases permeability of the blood vessel—an important characteristic that can help wound-healing substances in the blood reach injuries more efficiently.
As the world fights the SARS-CoV-2 virus causing the COVID-19 pandemic, another group of dangerous pathogens looms in the background. The threat of antibiotic-resistant bacteria has been growing for years and appears to be getting worse. If COVID-19 taught us one thing, it’s that governments should be prepared for more global public health crises, and that includes finding new ways to combat rogue bacteria that are becoming resistant to commonly used drugs.
In contrast to the current pandemic, viruses may be be the heroes of the next epidemic rather than the villains. Scientists have shown that viruses could be great weapons against bacteria that are resistant to antibiotics.
I am a biotechnology and policy expert focused on understanding how personal genetic and biological information can improve human health. Every person interacts intimately with a unique assortment of viruses and bacteria, and by deciphering these complex relationships we can better treat infectious diseases caused by antibiotic-resistant bacteria.
The hematopoietic (blood-forming) stem cells (HSCs) residing in our bone marrow produce all of our blood cells, including key immune cells that protect us from bacteria and viruses. As we age, our HSCs become less efficient and less able to make healthy new blood cells. In a study published online today in Nature, researchers at Albert Einstein College of Medicine have found that this reduction in HSC efficiency is caused in part by the deterioration of chaperone-mediated autophagy (CMA), the housekeeping process that removes damaged proteins and other waste materials that interfere with cells’ ability to function.
“While the aging of HSCs in our bone marrow is inevitable, the good news is that it may be reversible,” said co-study leader Ana Maria Cuervo, M.D., Ph.D., professor of developmental and molecular biology, of anatomy and structural biology, and of medicine, and the Robert and Renée Belfer Chair for the Study of Neurodegenerative Diseases at Einstein. “Our studies in mice suggest that drugs we’ve developed at Einstein can activate CMA and potentially restore the vitality of HSCs in older people.”
