A new Artificial Intelligence model manages to do complex physics simulations in real time with only using a fraction of the power that a traditionally computed simulation would use. These simulations could soon be used for things like biotechnology, gaming, weather predictions and more. Two Minute Papers has done several videos on it before, but this is a more complex AI with a wider range of applications. – TIMESTAMPS: 00:00 The Future of Advanced Physics Simulations. 01:57 How this new approach to AI works. 04:03 Are medical simulations a possibility? 06:02 Last Words. – #ai #physics #simulation
The Seoul Metropolitan Government (SMG) is the first local government in Korea to establish a metaverse platform, which has emerged as a contactless communication channel in the post-pandemic era, to start providing a new-concept public service by using the platform in its administration.
The SMG plans to establish “Metaverse Seoul” (tentatively named), a high-performance platform, by the end of next year, and create a metaverse ecosystem for all areas of its municipal administration, such as economic, cultural, tourism, educational and civic service, in three stages from next year.
Starting with the pilot program of a Bosingak Belfry virtual bell ringing event at the end of this year, the SMG will consecutively provide various business support facilities and services, including the Virtual Mayor’s Office, Seoul FinTech Lab, Invest Seoul and Seoul Campus Town, on its metaverse platform.
Sensors introduce an important new method to spot bio-marker for brain diseases Accurate timings of when brain signals fire demonstrated for the first time by the Sussex scientists, which has implications for tracking the onset of brain disease The quantum brain sensors could present a more efficient and accurate alternative to EEG and fMRI scanners.
In the future, soft robotic hands with advanced sensors could help diagnose and care for patients or act as more lifelike prostheses.
But one roadblock to encoding soft robotic hands with human-like sensing capabilities and dexterity has been the stretchability of pressure sensors. Although pressure sensors—needed for a robotic hand to grasp and pick up an object, or even take a pulse from a wrist—have been able to bend or stretch, their performance has been significantly affected by such movement.
Researchers at the Pritzker School of Molecular Engineering (PME) at the University of Chicago have found a way to address this issue and have designed a new pressure sensor that can be stretched up to 50 percent while maintaining almost the same sensing performance. It is also sensitive enough to sense the pressure of a small piece of paper, and it can respond to pressures almost instantaneously.
Scientists in South Africa are warning of a new strain of COVID-19. The variant, which is yet to be named, appears to have a high number of mutations, and there’s a possibility it could be able to evade our immune response and be even more transmissible. At first, health officials thought they were seeing a small cluster of outbreaks in South Africa’s most populous province. But after examining specimens, they realized they were dealing with something far more serious — a new variant that could be the hardest yet to contain. Officials are worried that the new variant, known simply as B.1.1.529, could quickly spread through the country and beyond. Only about 35% of adults in South Africa are fully vaccinated, and the rate of vaccination has slowed. And given the findings so far, even current vaccines may not be enough to stop it. Several countries, including the UK and Germany have announced a ban on flights from South Africa and five neighboring countries as cases of the new variant have already appeared in Botswana and in Hong Kong. No matter where the variant started, it could quickly become a global problem.
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The National Institutes of Health has launched a program to study a rare type of cells, called “senescent” cells, that play both positive and negative roles in biological processes. The NIH Common Fund’s Cellular Senescence Network (SenNet) program will leverage recent advances in studying individual cells, or single-cell analysis, to comprehensively identify and characterize the differences in senescent cells across the body, across various states of human health, and across the lifespan. The rarity and diversity of these cells previously made them difficult to identify and study; therefore, a deeper understanding will help researchers develop therapies that encourage beneficial effects of senescent cells while suppressing their tissue-damaging effects.
“The number of senescent cells in a person’s body increases with age, which may reflect both an increase in the generation of these cells and a decreased ability of the aging immune system to regulate or eliminate these cells. This age-related accumulation of senescent cells leads to production of inflammatory molecules and corruption of healthy cells,” said Richard J. Hodes, M.D., director of the National Institute on Aging, part of NIH. “This can affect a person’s ability to withstand stress or illness, recuperate from injuries, and maintain normal brain function. The aim of NIH’s strengthened focus on this field of science is to one day conquer these and other challenges.”
A cell dividing into two cells is a hallmark of human development. Over time, our bodies accumulate a small number of cells that no longer divide. These “senescent” cells can play important roles in health, either directly or through the release of molecules that affect neighboring cells. Senescent cells can play positive roles, such as aiding wound repair or preventing tumor growth in some cancers. However, they can also contribute to chronic diseases of aging such as cardiovascular disease and neurodegeneration. For this reason, therapeutics called “senolytics” are being developed to target senescent cells and remove them from the body.
We already have technology that allows wildlife to pass vaccines among themselves, and developing methods may speed up the process. But critics claim that they risk human infections and criminal misuse.
Scientists have discovered a way to stop the COVID-19 virus from replicating in infected human cells, marking major progress towards a definitive treatment for the deadly illness and accentuating the potential of genetic engineering to cure viral diseases.
The study explores the use of CRISPR, a genome editing tool, and builds on research that started at Australia’s Peter MacCallum Cancer Center in 2019, when Dr. Mohamed Fareh and Prof. Joe Trapani showed that CRISPR could be used to eliminate abnormal RNAs that drive children’s cancers.
At the beginning of the pandemic, and in collaboration with Director Prof. Sharon Lewin and Dr. Wei Zhao from the Doherty Institute, the scientists reprogrammed the same CRISPR tool to suppress replication of the RNA virus SARS-CoV-2 — and importantly, its “variants of concern” — in a test tube model. SARS-CoV-2, which is short for Severe Acute Respiratory Syndrome Coronavirus 2, is the virus that causes COVID-19.
Researchers have imaged a major component in conjugation—the process bacteria use to share DNA with each other.
During conjugation, bacteria can exchange genetic information in the form of special pieces of DNA. These include genes that help them resist attacks from common antimicrobial drugs, making many illnesses caused by these bacteria resistant to treatment.
Better understanding conjugation could therefore allow scientists to find ways to stop the process and reduce the spread of antimicrobial resistance.
