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The pace of replacing humans with robots in industries across China has been accelerating rapidly in the past couple of years, with observations on the ground suggesting that most industrial robotics and intelligent-manufacturing integrated service companies had at least doubled their annual sales in 2021.


Pandemic-led manufacturing export boom, concerns over China’s rapidly ageing society and a desire to save money have all contributed to the trend of replacing workers with machines.

The fascinating find shows how little is known about the ocean depths.

The vast colony, believed to be the world’s largest, is home to the remarkable icefish (Neopagetopsis ionah), which has a see-through skull and transparent blood. Icefish are the only vertebrates to have no red blood cells.

I’ve posted some vids of her before. But here she says at 3:52 that she thinks stopping the aging process is farfetched.


Dr. Morgan Levine, a professor who specializes in the biology of aging, answers the internet’s burning questions about aging. Is there anyway to stop aging? Is aging a disease? Do you age slower in space? Dr. Levine answers all these questions and much more!

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What are epinutrients, you ask? Allow us to explain.


In a 2021 study, naturopathic doctor and functional medicine expert Kara Fitzgerald, N.D., author of Younger You, was able to improve participants’ biological age by three years. Sounds like a tall order, but the intervention was actually pretty simple: With a very doable diet and exercise plan, it only took eight weeks to see these results. “A big reason we actually made a difference in eight weeks’ time is because we very intentionally bathed the body in a high amount of epinutrients,” she says on this episode of the mindbodygreen podcast.

What are epinutrients, you ask? Allow Fitzgerald to explain below, along with a few of her favorites.

Gene editing approaches promise to treat a range of diseases, but delivering editing agents to cells in animal models and humans safely and efficiently has proven challenging. Now, researchers led by a team at the Broad Institute of MIT and Harvard have developed a way to get gene editing proteins inside cells in animal models with high enough efficiency to show therapeutic benefit.

In new work published in Cell, the team shows how they have engineered virus-like particles to deliver base editors — proteins that make programmable single-letter changes in DNA — and CRISPR-Cas9 nuclease, a protein that cuts DNA at targeted sites in the genome. In collaboration with ​​research teams led by Krzysztof Palczewski at the University of California, Irvine, and Kiran Musunuru at the Perelman School of Medicine at the University of Pennsylvania, the team used their particles, called engineered virus-like particles (eVLPs), to disable a gene in mice that can be associated with high cholesterol levels, and partially restored visual function to mice harboring a mutation that causes genetic blindness.


Researchers have developed virus-like particles that deliver therapeutic levels of protein to animal models of disease.

In a new study, researchers at Karolinska Institutet have identified the presence of a specific connection between a protein and an lncRNA molecule in liver cancer. By increasing the presence of the lncRNA molecule, the fat depots of the tumor cell decrease, which causes the division of tumor cells to cease, and they eventually die. The study, published in the journal Gut, contributes to increased knowledge that can add to a better diagnosis and future cancer treatments.

Bioelectricity, the current that flows between our cells, is fundamental to our ability to think and talk and walk.

In addition, there is a growing body of evidence that recording and altering the bioelectric fields of cells and tissue plays a vital role in and even potentially fighting diseases like cancer and heart disease.

Now, for the first time, researchers at the USC Viterbi School of Engineering have created a molecular device that can do both: Record and manipulate its surrounding bioelectric field.