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A team of scientists recently revealed they’d successfully conducted experiments on hundreds of pigs that involved keeping their brains alive for up to 36 hours after the animals had been decapitated.

Maybe Sergio Canavero, the mad scientist who wants to perform a brain transplant on a human, isn’t so crazy after all.

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Scientists have identified special types of brain cells that may allow us to simulate the decision-making processes of others, thereby reconstructing their state of mind and predicting their intentions. Dysfunction in these ‘simulation neurons’ may help explain difficulties with social interactions in conditions such as autism and social anxiety.

Researchers at the University of Cambridge identified the previously-unknown neuron type, which they say actively and spontaneously simulates mental decision processes when social partners learn from one another.

The study, published today in Cell, suggests that these newly-termed ‘simulation neurons’ — found in the amygdala, a collection of nerve cells in the temporal lobe of the brain — allow animals (and potentially also humans) to reconstruct their social partner’s state of mind and thereby predict their intentions.

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Stimulating the brains of elderly people with electrical currents allowed them to perform just as well on a memory test as people in their 20s — a sign that researchers may have found a noninvasive way to turn back the hands of time when it comes to human memory.

“It’s opening up a whole new avenue of potential research and treatment options,” researcher Rob Reinhart said in a press release regarding the study, “and we’re super excited about it.”

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Scientists say they’ve developed a new “quantum material” that could one day transfer information directly from human brains to a computer.

The research is in early stages, but it invokes ideas like uploading brains to the cloud or hooking people up to a computer to track deep health metrics — concepts that until now existed solely in science fiction.

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‘’Examining how deep disagreements arise will demonstrate the gravity of the issue. Why do we disagree with valid, knowable facts when we all live in the same world, we have roughly the same cognitive abilities and, in the Western world at least, most people have fairly easy access to roughly the same information?


What happens when you can’t agree on the facts?

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Researchers may be closing in on a way to check athletes while they’re alive for signs of a degenerative brain disease that’s been linked to frequent head blows. Experimental scans found higher levels of an abnormal protein tied to the disease in a study of former National Football League players who were having mood and thinking problems.

It’s the first time a major study has tested these scans for detecting chronic traumatic encephalopathy, or CTE, which is only diagnosed now after death, with brain autopsies.

Doctors are searching for a way to tell when players, veterans or others with concussions or other head injuries are at risk for permanent damage. It’s too soon to know if the scans will enable that — so far they only show that these athletes are different as a group; they can’t be used to say a particular player does or does not have CTE.

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Transforming the microbial environment in the guts of children diagnosed with autism could significantly ease the severity of their condition’s signature traits, according to newly published research.

A study on the effects of a form of faecal transplant therapy in children on the autism spectrum found participants not only experienced fewer gut problems, but continued to show ongoing improvements in autism symptoms two years after the procedure.

Arizona State University researchers had already discovered a dose of healthy gut microflora caused characteristics associated with autism spectrum disorder (ASD) to ease or vanish for at least a couple of months after treatment ended.

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But not everyone is on board.

“The use of transgenic monkeys to study human genes linked to brain evolution is a very risky road to take,” University of Colorado geneticist James Sikela told the MIT Technology Review. “It is a classic slippery slope issue and one that we can expect to recur as this type of research is pursued.”

Pinpointing the gene’s role in intelligence could help scientists understand how humans evolved to be so smart, MIT Tech reports.

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In a stunning piece of research, Stanford neuroscientists have hunted down a single gene that encodes a protein responsible for age-related cognitive losses, targeted it with special blocking antibodies, and shown in mice that these antibodies can rejuvenate old brains to work as well as young ones.

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