Lifeboat Foundation: Safeguarding Humanity
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Category: biotech/medical

CHICAGO — Small RNA molecules originally developed as a tool to study gene function trigger a mechanism hidden in every cell that forces the cell to commit suicide, reports a new Northwestern Medicine study, the first to identify molecules to trigger a fail-safe mechanism that may protect us from cancer.

The mechanism — RNA suicide molecules — can potentially be developed into a novel form of cancer therapy, the study authors said.

Cancer cells treated with the RNA molecules never become resistant to them because they simultaneously eliminate multiple genes that cancer cells need for survival.

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A research team at MIT has used synthetic biology to create a gene circuit that triggers the immune system to attack cancer when it first detects the signs of the disease.

The circuit works by only activating the immune response when two specific cancer biomarkers are detected. The new study was published in the journal Cell this week and represents an exciting step forward for synthetic biology and cancer research.

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ProPublica has been documenting the many ways health care dollars are being wasted. We’ve shown how hospitals throw out brand new supplies, nursing homes flush tons of unexpired medication and drug companies concoct costly combinations of cheap medication. Recently we described how arbitrary drug expiration dates cause us to toss safe and potent medicine.

Often, large swaths of the medical and pharmaceutical communities know about this waste — even about solutions to it — but do nothing. Those who end up paying the bill, in one way or another, are consumers.

The makers of cancer drugs also make vials with too much medication for many patients. The excess drugs are tossed in the trash — another reason health care costs are so high.

By Marshall Allen

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Can the process of aging be delayed or even reversed? Research led by specially appointed Professor Jun-Ichi Hayashi from the University of Tsukuba in Japan has shown that, in human cell lines at least, it can. They also found that the regulation of two genes involved with the production of glycine, the smallest and simplest amino acid, is partly responsible for some of the characteristics of aging.

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Last year, Dr. Sergio Canavero created quite the ruckus (to put it mildly) when he vowed to be the first person to transplant a human head onto a deceased donor’s body. Yes, he is planning on attempting the world’s first human head transplant (or body transplant, depending on how you look at it).

In fact, it has been about a year since his initial proclamation, and the Italian neurosurgeon still stands firm on his declaration, despite claims from other experts that it is nothing but a PR Stunt (at best) or a hoax. Some have even hypothesized it’s all just a plot meant to promote Metal Gear Solid.

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LEF has access to blood tests from its customers who take the product. That means data should be available in less than a year. If it works, we can expect other DNN-developed geroprotectors.

In 2011, scientists made one of the most important discoveries in the history of AI development. They found that graphics processing units (GPUs) are far better at simulating biological learning than central processing units (CPUs).

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If scientists could precisely regulate gene expression, they could turn off the genes responsible for illness and disease and turn on those that enhance health and the immune system.

“This is why controlling gene expression is so fundamental,” said Northwestern University’s Julius Lucks. “Once you get a good handle on it, you can do anything.”

For Lucks, having a “good” handle on might be an understatement. He and his team have developed a powerful and versatile tool that achieves gene activation thousands of times better than nature.

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